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  • Smad2 Mediates Transforming Growth Factor-β Induction of Endothelial Nitric Oxide Synthase Expression

  • Nitric Oxide Regulates Transforming Growth Factor-β Signaling in Endothelial Cells

  • Abstract 12: EMMPRIN-Targeted Magnetic Nanoparticles for in vivo Visualization and Regression of Acute Myocardial Infarction in a Model of Acute Coronary Artery Occlusion

  • Abstract 620: Circulating miRNAs a Novel Tool to Assess BAG3 Related Dilated Cardiomyopathy

  • H2O2 Regulation of Vascular Function Through sGC mRNA Stabilization by HuR

  • Nitric Oxide Prevents Aortic Neointimal Hyperplasia by Controlling Macrophage Polarization

  • iNOS-Derived Nitric Oxide Induces Integrin-Linked Kinase Endocytic Lysosome-Mediated Degradation in the Vascular Endothelium

  • Abstract 19684: Nitric Oxide Protects From Cardiac Ischemia/reperfusion Damage by Blocking Activation of Extracellular Matrix Metalloproteinase Inducer EMMPRIN Through Binding to Caveolin3

  • Abstract 11738: Targeting Endothelial Integrin Linked Kinase Induces Spontaneous Myocardial Infarction and Regulates Cardiac Remodeling

  • Abstract 12255: Differential Expression of miRNAs in BAG3-mutation Carriers With Dilated Cardiomyopathy

  • Abstract 13438: Specific Circulating Microparticle Components Secreted in Heart Failure Are New Targets of Ivabradine

  • Abstract 14598: Ivabradine Induces Cardiac Protection by Preventing Inflammatory Response Through Inhibition of Cyclophilin-A Secretion After Myocardial Ischemia/Reperfusion Injury

  • Abstract 14758: Deletion of Endothelial Integrin Linked Kinase (ILK) Accelerates the Development of Calcific Aortic Valve Disease

  • Abstract 13243: Integrin Linked Kinase Expression in Human Valvular Endothelial Cells Plays a Protective Role in Calcific Aortic Valve Disease

  • Theranostic Contribution of Extracellular Matrix Metalloprotease Inducer-Paramagnetic Nanoparticles Against Acute Myocardial Infarction in a Pig Model of Coronary Ischemia-Reperfusion

  • Integrin-Linked Kinase Regulates Vasomotor Function by Preventing Endothelial Nitric Oxide Synthase Uncoupling: Role in Atherosclerosis